glp-1 secretion mechanism receptor signalling promotes β-cell glucose metabolism via mTOR-dependent HIF-1α activation The Actin Cytoskeleton and GLP-1

glp-1 secretion mechanism

glp-1 secretion mechanism receptor signalling promotes β-cell glucose metabolism via mTOR-dependent HIF-1α activation The Actin Cytoskeleton and GLP-1

Name: glp-1 secretion mechanism receptor signalling promotes β-cell glucose metabolism via mTOR-dependent HIF-1α activation The Actin Cytoskeleton and GLP-1
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glp-1 secretion mechanism receptor signalling promotes -cell glucose metabolism via mTOR-dependent HIF-1 activation The Actin Cytoskeleton and GLP-1

[DOI] [PMC free article] [PubMed] [Google Scholar] 313.Allegretti JR, Mullish BH, Kelly C, Fischer M

How It Works High signal amplification in the cell Strong efficacy of receptor-effector coupling Common in tissues with efficient signaling cascades (like heart and smooth muscle) Example: -adrenoceptors in cardiac muscle, only a small proportion need to be activated for full contractile response