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Since the stigma measures were associated with smoking cessation efficacy and controllability of smoking, they were included as covariates (along with the other significant correlates found) in the models used to explore Aims 2 and 3 (determining the relationships between the stigma measures, scenario, and lung cancer concern/perceived risk)

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One popular explanation [6, 69] is that smoking alters catecholamine release, and hence free fatty acid release, which in turn affects VLDL and LDL concentrations to favour their accumulation in blood, contributing to a lower HDL concentration