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In contrast to genetic mutations, epigenetic modifications rapidly affect target cell types exerting a long-lasting control in gene expression and provide a mechanism for a non-genomic inheritance
Gene set enrichment analysis (GSEA) confirmed the association between GSH metabolism and MTX resistance
1b) [26]
While a fibrotic matrix initially contributes to tissue repair, after injury, chronic and persistent kidney damage in the development of CKD leads to uncontrolled fibrotic matrix deposition, resulting in reduced blood supply, destruction of normal organ structure, and eventual glomerulosclerosis, renal tubular atrophy, and interstitial fibrosis [9, 10]
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