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Employing gene editing techniques alongside both in vivo and in vitro experiments, we provide novel evidence that the dysfunction of SLC25A39-mediated mitochondrial import of GSH constitutes a pathological alteration following TBI, leading to mitochondrial dysfunction and subsequent initiation of oxidative stress and neuronal cell death

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Moreover, ionizing radiation can induce pyroptosis in cancer cells expressing the GSDME protein, such as those found in lung, liver, breast, and glioma cancers [189]