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All such forward-looking statements speak only as of the date of this press release/company announcement and are based on information available to Zealand Pharma as of the date of this release/announcement

Experimental evidence from rats exposed to early postnatal (days 5-16) treatment with l-buthionine-(S,R)-sulfoximine (BSO), an accepted rodent model of oxidative stress which induces a transitory deficit in GSH, have shown that GSH deficiency leads to long-lasting behavioral aberrations (Cabungcal et al

Dietary tryptophan restriction in EAE models can abolish BBB disruption, leukocyte infiltration, and CNS demyelination, likely by inhibiting Th1/Th17 skewing and impairing migratory capacity (311)

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