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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial
slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial

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(PubMed) Moses EK, Johnson MP, Tommerdal L, et al

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial

Taken together, our data indicate that the loss of GSH triggered by Gclc deletion induces a defective mitochondrial accumulation in FoB, which may lead to compensation through upregulation of glycolytic metabolism

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial

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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial

however, in the cerebral cortex, only Se-deficient diet led to iron accumulation (Sharma et al., 2019)

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Autoregulatory control of homeostasis SLC25A39 is necessary for mitochondrial
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